0:01

Many cases of sudden death and

0:03

severe disease are being reported since

0:06

the rollout of the COVID-19

0:14

gene-based vaccines. Early

0:17

on, several doctors and scientists

0:20

hypothesized that the COVID vaccines

0:22

would lead to several complications

0:25

including autoimmune disease, blood clots,

0:27

strokes, and more. Additionally,

0:30

the Vaccine Adverse Event Reporting

0:32

System, or VAERS data, showed

0:34

a strong correlation between the

0:36

vaccines and adverse events. The

0:40

warning signs were always there, but

0:42

most of the evidence that is

0:44

discussed surrounding adverse events is focused

0:46

on the numbers. This

0:49

many more sudden deaths, stillbirths,

0:51

or cases of myocarditis, for

0:53

example. So

0:55

how does one determine, in an

0:57

individual case, that the vaccine was

0:59

the cause of death or the

1:01

adverse event? It

1:04

is done through pathology. An

1:06

early pioneer of pathological investigations

1:09

into vaccine adverse events

1:11

was Professor Arna Burkhardt,

1:13

a senior, highly accomplished

1:15

pathologist from Germany. Professor

1:18

Burkhardt came out of retirement

1:20

in 2021 to examine the

1:23

autopsy and biopsy materials of

1:25

vaccinated patients both living and

1:27

deceased. The

1:29

work of Professor Burkhardt not

1:31

only provided strong evidence of

1:33

vaccine causation, it substantiated

1:36

the professional medical hypotheses

1:39

of doctors and scientists around the

1:41

world. Unfortunately,

1:44

in May 2023, Professor Burkhardt passed away.

1:54

But shortly before his death, I had

1:56

the chance to interview him in his

1:58

laboratory in Germany. in which

2:00

he gave a detailed and compelling account

2:03

of his work. During

2:06

the next two hours, you

2:08

will hear Professor Burkhart once

2:10

more in his own words,

2:12

discuss his findings, his motivation,

2:15

and what he hoped for the future of

2:17

the fields of science and medicine. This

2:20

is one of the few extensive

2:23

English language interviews with Professor Arna

2:25

Burkhart, and it is one

2:27

of his last. I'm

3:01

journalist Taylor Hudak, and today I'm

3:03

in Roichlingen, Germany, at the Laboratory

3:05

of Pathologists Professor Dr. Arna Burkhart.

3:08

Professor Burkhart is a highly esteemed pathologist

3:11

with more than 50 years experience

3:13

in the field. Since 2021, he

3:16

has examined 75 autopsies in patients

3:18

who died shortly after vaccination, as

3:21

well as 41 biopsies

3:23

in living persons, to determine

3:25

if the COVID-19 vaccinations cause

3:27

either the deaths or disease

3:29

in the patient. All

3:32

right, Professor Arna Burkhart, it is my pleasure

3:34

to be here in your lab today

3:36

and to speak with you. I have

3:38

been following your work very closely for

3:40

the past year in particular. So

3:43

why don't you just introduce yourself to

3:45

the viewers, explain your credentials, your qualifications,

3:47

as well as your contributions to the

3:50

field of pathology. Well,

3:52

first of all, of course, I would like

3:54

to thank you for the opportunity to talk

3:57

to you and for those people.

4:00

people who are interested in this field.

4:03

Well, as you said, I've

4:06

been in the field of

4:08

pathology for many, many years

4:10

now and many

4:13

years also in scientific projects

4:16

involved. The latest

4:18

book has appeared in just

4:20

the last year, so I

4:22

think I'm still in

4:24

the business, so to say. Actually

4:27

beginning of 2021, I wanted to

4:30

close this laboratory and go into

4:32

retirement. And

4:38

just at that moment, the

4:41

vaccination campaign started in Germany

4:43

and it only took three

4:45

months. That was

4:48

in March 21 that

4:50

the first reports came to me

4:52

about serious side effects and especially

4:57

cases of people who died in timely

5:00

connection with the vaccination. And

5:04

in most of these cases, even

5:07

if an autopsy was

5:09

performed, it was stated well this

5:12

was a natural death and the

5:14

relatives were suspicious about this

5:17

and they didn't accept this

5:19

because usually these

5:21

people were very healthy before

5:23

vaccination. So they

5:26

contacted me and other

5:28

pathologists about a second

5:30

opinion. Now a

5:32

second opinion is something very usual

5:35

in oncology because to

5:38

type a certain type

5:40

of cancer, you have

5:43

many, sometimes many pathologists

5:45

look at it and they have

5:47

different opinions and so on. But

5:50

in autopsy, usually the autopsy is

5:53

considered something like a gold standard.

5:56

If you do the autopsy and you have a

5:58

result, you accept it. Maybe

6:02

only once a year it happened that somebody

6:04

said, well, there is an out of sea

6:06

and I don't believe the results, please look

6:09

at it. But suddenly

6:11

these were many, many relatives

6:14

that came to me and

6:16

sometimes also attorneys

6:19

who turned to me and asked

6:21

if I would be

6:23

willing to do this, because usually

6:26

many other pathologists

6:28

just refused this. And

6:32

actually I said, well,

6:34

I can look at five or

6:36

six cases and probably

6:38

everything is okay and this will

6:40

be it. But

6:43

then I received the first

6:46

five cases and I saw

6:48

things that were very unusual.

6:51

And reasons that I

6:54

had not seen before in

6:56

this context. So

6:59

actually I contacted

7:02

other pathologists and

7:04

also university

7:07

institutes and asked them

7:09

if they would continue

7:11

this work and take

7:13

over my project, because

7:15

actually I didn't want

7:17

to go into retirement.

7:21

But actually some

7:23

of the pathologists

7:26

that I contacted first said, well,

7:28

yes, yes, we do it and

7:30

we have some support from the government for

7:32

these projects and we will do it.

7:35

But after a while when it

7:38

should have been started, they

7:41

drew back and they said, we don't

7:43

want to have anything to do with

7:45

it, please leave us alone. So

7:48

I was forced

7:50

to continue my work and

7:54

as a consequence of our first

7:58

results, Luckily

8:00

I had a second experience,

8:03

pathologist Professor Lang from Binova,

8:06

who helped me and who

8:08

was able to confirm

8:11

what I saw. Of

8:13

course the initial diagnosis was

8:16

either death caused by some natural

8:18

causes or another cause.

8:20

So how do you account for

8:22

the discrepancy between your second opinion

8:24

findings and the initial cause of

8:27

death? Well

8:29

as you said there was

8:31

a discrepancy in almost

8:34

all of the cases that we saw. The

8:38

pathologist or the corona who

8:40

did the first autopsy claimed

8:43

it was a natural cause of

8:45

death or some stated

8:48

it was unclear. Okay that's

8:50

always a very honest diagnosis.

8:53

The problem is that quality

8:56

of autopsy as

8:58

it is practiced now in Germany

9:00

I think has dramatically

9:02

declined in the last years. When

9:05

I learned pathology the

9:08

main focus of the Institute of

9:10

Pathology was autopsy but

9:12

now it's mostly bioptic

9:14

diagnostic which of course

9:17

is also very important but

9:20

pathologists have lost

9:23

the interest in autopsy and

9:25

usually they

9:28

are satisfied if they find something

9:30

plausible as a death. So if they

9:32

see a discoloration of the heart

9:34

muscle they say well this

9:36

is an infarction of

9:39

the heart and if the person is

9:41

older than 50 years I

9:44

mean that's always plausible. So

9:50

they no longer look for the

9:53

causes behind what they see.

10:00

In many cases histology is not done,

10:02

that means the tissue is

10:04

not examined in the microscope and

10:06

you cannot make a diagnosis without,

10:10

or not many diagnosis you

10:12

cannot make without looking

10:14

at the microscope. And that's what

10:16

you did, you used histopathology, is that

10:18

correct? Yes. And can you explain to

10:21

us what that is? We will also

10:23

show these images here to help

10:25

explain the use of histopathology.

10:29

Yes, well first of all you

10:32

take a small specimen

10:35

from the tissue that you want to

10:37

examine and it has

10:39

to be fixed because it's soft and

10:41

you cannot cut it and you have

10:43

to perform

10:47

very thin sections, thousands

10:50

of millimeters of thickness and

10:52

if you put them on a glass

10:54

slide so they are fixed then

10:58

they don't have any color at all. Only

11:01

few elements have color like the red

11:03

blood cells, that's why they are called

11:05

red blood cells but all the other

11:07

cells do not have

11:09

any color. So you

11:12

use special stains to

11:15

make structures with

11:17

them and there are two

11:19

ways to do this. One

11:22

is that you have a

11:24

chemical affinity of the dye

11:26

and you see special structures

11:29

and the other thing is that

11:32

you have antibodies that bind to

11:34

certain proteins that you can see

11:36

in what we call immunohistochemistry. Let's

11:39

focus on this image here, this

11:42

explains on the left or it

11:44

shows rather unstained prostate gland tissue

11:46

and kidney tissue and then on

11:48

the right we see it labeled

11:50

as H-E stain. Can you just

11:53

explain why you would use this

11:55

method when examining a specimen? Well

11:58

actually the H and E stain

12:00

which is hematoxylen in

12:57

addition to the HE stain, there

12:59

are also various special staining methods

13:01

that highlight specific

13:24

structures and disease related

13:26

features. Here we see

13:28

two examples from Professor Burkhardt's findings.

13:31

In the image on the left, the Congo

13:33

Red stain is used to highlight a ring

13:36

of amyloid within the vessel, which is a

13:38

darker red color. In the

13:40

image on the right, the Prussian Blue

13:42

stain is used to highlight iron deposits

13:44

in the periphery of the vessel. Later

13:47

on in this interview, Professor Burkhardt

13:50

will explain what the findings

13:52

mean. I next asked

13:54

him whether the pathologists who

13:56

had performed the initial autopsies

13:58

had also examined the tissue

14:00

samples under the microscope. Those

14:04

autopsies that were performed in

14:08

legal institutions, usually

14:12

no histology at all

14:14

is done. Is that unusual to

14:16

you? No, this

14:19

has been practiced since

14:21

the beginning of legal medicine, actually,

14:24

and it is upon

14:27

the prosecutor if he orders histology

14:29

to be done or not. So

14:32

of course, if there's a clear case, like

14:35

somebody has been stabbed

14:37

by a knife, I mean, you don't

14:39

have to have a histology for that.

14:41

And so the prosecutor says, well, this

14:43

is okay. But if

14:46

it is stated unclear, of

14:48

course, if this is unclear,

14:51

then the prosecutor has to decide

14:53

whether histology is done or not.

14:56

But actually, in many

14:58

cases that I have seen

15:00

now, even if the coroner

15:02

said cause of death is

15:04

unclear, no histology was ordered. In

15:06

many cases, toxicological examinations

15:09

were done, especially in young

15:12

people who die suddenly of

15:14

unexplained causes. Often

15:17

drug abuse is suspected.

15:21

But in all of these cases that

15:23

we examined, this was negative. So

15:26

in your own studies, upon your second

15:28

opinion, you did use the

15:31

histopathology, whereas it was not

15:33

done as an initial diagnosis.

15:36

Yes, yes. So that accounts for

15:38

the discrepancy in your second opinion compared

15:40

to the initial diagnosis. Usually,

15:42

yes. As I said,

15:44

the legal

15:47

institutions do not

15:49

histology as

15:51

a routine work, but they

15:53

preserve specimens. And these

15:56

specimens are guarded for two

15:58

or three years. So we

16:00

got these specimens and we

16:03

did the histology. Now

16:05

I would like to focus on

16:08

the pathological changes commonly seen in

16:10

vaccinated persons. And so many of

16:12

us who are not medical professionals

16:14

have heard of myocarditis, which is

16:17

inflammation of the heart muscle tissue.

16:19

And you were able to observe this in

16:22

your own studies. So I would like to

16:24

now discuss these two images here. This

16:27

shows on the left normal heart

16:29

muscle tissue and on

16:31

the right lymphocytic myocarditis after the

16:34

vaccine. Can you explain to us

16:36

the observed abnormalities that you see with

16:38

the image on the right? Well,

16:41

on the left side, you see the muscle

16:44

cells, which are elongated and

16:46

have these long nuclei.

16:48

But on the right side, you see

16:50

that in

16:53

the middle between these muscle

16:55

cells are small blue

16:57

dots, which are the

17:00

nuclei of lymphocytes. Lymphocytes

17:02

are immunologically active

17:04

cells. And apparently they have

17:06

been attracted by some antigenic

17:10

material that is in the heart.

17:13

Now, of course, one

17:15

or two lymphocytes are

17:18

always seen in a section,

17:20

but not aggregations like that.

17:24

You may compare this to the police

17:27

controlling city, for

17:29

example. Now, if you

17:31

see one policeman or a policewoman,

17:35

that's okay. I mean, that's normal.

17:37

If you see two, it's

17:39

still not alarming. But if you all

17:42

of a sudden you see 50 policemen,

17:45

then you know there must be some trouble

17:47

somewhere in the city. And that's the same

17:49

in the heart muscle. I mean, if

17:52

I see one or two lymphocytes,

17:54

that's normal. And they control,

17:56

so to say, if there's anything wrong.

17:59

But... If they are aggregated

18:01

like this in clumps, there's

18:04

something wrong and that's myocarditis.

18:06

By the way, myocarditis, as

18:09

a consequence of vaccination, of

18:12

the so-called vaccination, is

18:14

now internationally recognized. I mean, this

18:17

is nothing that we have to

18:19

prove anymore. This has been proven

18:22

and is a solid

18:24

scientific standard. Absolutely.

18:27

Now with this particular

18:29

case that we are referencing here, do you know what

18:31

symptoms the person had? And if

18:33

not, what symptoms would you expect one to

18:35

experience in a case like this? Well,

18:39

the main symptom is fatigue and

18:46

physical deterioration, the necessity to sleep

18:49

after some sportive activity and

18:54

physical strain. So

18:57

actually, we had 31 cases

19:00

among the 75 cases where

19:07

it was stated that they died

19:09

of heart failure and

19:11

normal heart failure like rifbot, genetic

19:14

failure, and so on. And

19:17

of these 31 cases, actually in 15 cases, there

19:22

was a perimuocarditis

19:25

inflammation. And in the other

19:27

16, we saw what

19:29

is called a microangiopathy that

19:31

is changes in the small

19:33

vessels that have nothing

19:36

to do with arterial sclerosis, which

19:39

of course is normal in older patients. So

19:41

this is the muocarditis.

19:47

Prior to the rollout of the

19:49

COVID-19 vaccinations, when you look back

19:51

on your career, how often did

19:53

you see myocarditis then compared to

19:56

now within the past two years?

20:00

I don't think I saw buocarditis

20:02

more than once a year. And

20:05

at that time we did between

20:08

1,500 and 2,000 autopsies a

20:13

year, and as

20:15

I said, one or two cases a year.

20:18

And now this is one of

20:20

the most common diagnosis especially in

20:23

younger people. Professor

20:25

Burkhart has explained that clusters

20:27

of lymphocytes in heart muscle

20:30

tissue indicate inflammation. A

20:33

large cluster of lymphocytes is also

20:35

seen in this sample of lung

20:37

tissue. The sample is from an

20:39

82 year old woman who died

20:41

40 days after receiving

20:43

a second injection of the Moderna

20:46

vaccine. You

20:48

can see a small vessel

20:51

and there's a

20:53

lymphocytic infiltration around it which

20:55

is not normally in the

20:57

lung. And

20:59

so this person definitely

21:02

must have had some deficit

21:05

in the gas exchange of the lung.

21:08

We next discuss the case of

21:10

a 70 year old woman in

21:12

whom Professor Burkhart found striking changes

21:14

within the thyroid gland. A

21:17

tissue sample from the woman's thyroid gland

21:19

is shown on the right. On

21:22

the left, we see some normal

21:24

thyroid gland tissue for comparison. Well,

21:27

yes, on the left side, you see

21:29

the, what

21:31

we call follicles, which

21:33

contain the thyroid hormone.

21:35

And the hormone is of course

21:37

needed for the body. And

21:40

on the right side, you see

21:43

that these structures are lacking.

21:46

And instead of these

21:48

structures, there's lymphocytic infiltrations

21:52

which we already have seen in the

21:54

other pictures. So

21:56

these lymphocytes destroy the thyroid

21:59

gland. and

22:01

this is a well-known autoimmune disease,

22:04

which is known for many years

22:06

and it of course occurs without

22:08

a vaccination. But

22:11

after this vaccination, we

22:14

see it more often. And in

22:16

these people that we saw the

22:18

autopsies, in many cases this disease

22:20

was not known before. So it

22:23

probably started or was at

22:25

least promoted by the vaccination.

22:28

We asked about if there were

22:31

symptoms before and nobody knew anything

22:33

about a thyroid disease in this

22:35

testing. So this person did not have

22:37

any pre-existing condition that could maybe put them at risk

22:39

for something like this? No. Interesting.

22:42

Okay. Is this damage

22:45

reversible? Once the

22:47

thyroid GANS is destroyed,

22:49

this is not reversible. It does not

22:51

have the capability of

22:55

reconstruction. But

22:57

it can be treated of course

22:59

by giving the hormone medical treatment.

23:26

The mRNA binds to the ribosomes within the

23:28

cell, which are the cells little protein factories.

23:32

The ribosomes read the information on the

23:34

mRNA and create the DNA that is present

23:36

in the cell. The

23:45

mRNA binds to the ribosomes

23:47

within the cell, which are the

23:49

cells little protein factories. The

23:52

ribosomes read the information on

23:54

the mRNA and create multiple

23:56

copies of the spike protein

23:59

molecules intact. The exact spike

24:01

protein molecules will transfer to the

24:03

cell surface and some

24:05

spike protein molecules are fragmented

24:08

and the fragments are taken to the

24:10

cell surface. There they

24:12

are presented to the cells of the

24:14

immune system by a specific carrier

24:17

molecule called MHC1. MHC1

24:20

is the orange figure shown

24:22

here. Think of

24:24

MHC1 as a passport and

24:26

the antigenic peptide or the

24:29

spike protein fragment carried by

24:31

MHC1 as the individual details

24:33

printed within the passport such

24:35

as name and photograph. T

24:39

lymphocytes which happen to possess T

24:41

cell receptors which match these antigenic

24:43

peptides or spike protein fragments will

24:46

recognize the MHC1 in combination with

24:48

the spike protein fragments it carries

24:51

and then bind to it. If

24:54

a cytotoxic T cell recognizes

24:56

and binds its matching antigenic

24:58

peptide then it will attack

25:00

and destroy the cell which

25:02

presents it. This is

25:04

a necessary step in antiviral

25:07

defense. However, in

25:09

the context of vaccination it

25:11

is unnecessary and potentially dangerous

25:14

as the immune

25:16

system will attack healthy cells.

25:20

Lymphocytes occur in the spleen and lymph

25:22

nodes but are also seen in the

25:24

blood. The lymphocytes are

25:26

fairly small, are round and

25:28

are typically stained dark purple.

25:31

If lymphocytes appear in large quantities

25:33

in tissues other than the lymph

25:36

nodes or the spleen this usually

25:38

means that either a viral infection

25:40

or some autoimmune disease is in

25:43

progress. A third possibility would

25:45

be the rejection of a transplanted organ.

25:48

Now we must contemplate

25:50

another novel mechanism namely

25:52

the attack of the immune system

25:55

on the vaccine expressing cells. to

26:00

enter the bloodstream shortly after injections.

26:03

And this does raise questions as to how

26:05

it could impact the blood vessels. So what

26:07

have been your observations on this point? Well,

26:11

first of all, the spike protein,

26:13

which is on the one hand

26:15

produced by the virus in viral

26:17

detection, but on the other side

26:19

is induced by the vaccination in

26:21

the body. But these

26:24

two possibilities have completely different

26:26

access to the body cells.

26:29

If you have the

26:31

viral infection, the toxic

26:34

spike protein first

26:36

has to pass through the

26:39

epithelium. And the epithelium is

26:41

immunocompetent and already

26:43

has the capability of

26:46

detoxification and the destruction

26:48

of harmful elements.

26:51

But the endothelium,

26:54

which is the lining of

26:56

the vessels, is not

26:59

an immunocompetent cell structure.

27:02

So the toxin that is

27:04

entering into the bloodstream

27:07

and into the lymphatic vessels

27:10

directly hits the cells

27:12

that are not able

27:14

to defend themselves.

27:16

So they may be

27:19

destroyed, they may be destroyed by

27:21

toxic action, and they may be

27:23

destroyed by immunologic

27:25

attack. Blood vessels

27:27

are preferred targets of lymphocyte

27:30

attack after vaccination. Both

27:32

the large and small blood vessels

27:34

can be afflicted anywhere and everywhere

27:36

in the body. This is because

27:39

the vaccine will distribute from the

27:41

injection site to other locations in

27:43

the body, mainly through the bloodstream.

27:47

This image shows a dissection of the

27:49

aorta, the largest blood vessel in the

27:52

body. An aortic dissection

27:54

is usually very rare. It

27:56

is a serious condition in which a tear in the aorta

27:58

is caused by a blood vessel. allows blood

28:01

to rush into the vessel wall, causing

28:03

it to split or dissect. This

28:06

patient was a 55-year-old male

28:08

who died 21 days

28:10

after the second injection. What

28:13

is being observed here and why is it significant?

28:16

Can you talk about this case in particular? Yes,

28:19

you see a section of

28:22

the aorta and if

28:24

you see on the left side, there's

28:26

a solid wall, which

28:29

is a kind of yellow coloration.

28:31

Yellow is the color

28:33

of the elastic fibers, by the way.

28:36

And then you see that there's a split

28:38

formation in the middle and then

28:42

on the right side, there are actually

28:44

two walls. And in

28:46

the middle, there's this

28:49

black material which actually is blood.

28:51

So there has been blood flown

28:54

into this dissected

28:56

aorta. The media,

28:58

what we call the media of

29:00

the aorta has been destroyed and

29:02

the blood has entered. And once

29:05

the blood has entered there, then

29:07

the aorta may rupture

29:09

and the people die of

29:12

blood loss. I also

29:14

want to look at this image from

29:16

a microscopic viewpoint. So here again is

29:18

the dissection of the aorta. This

29:21

is the same aortic wall, but this time it is

29:23

stained with AT, which we talked about

29:25

earlier and placed under a microscope. So

29:28

here are the two images. Can

29:30

you describe in a greater detail what we

29:32

are seeing here now that

29:34

it's under the microscope and there's also

29:36

dye applied, what can we tell about

29:38

this aortic dissection? Yes, if you

29:40

look at the left side, on

29:43

top you see the lumen of

29:46

the aorta where once the blood

29:48

was flowing. Then there

29:50

is the inner section

29:53

of the aorta and

29:55

below there's a dissection and then you

29:58

have the... outer

30:00

wall. So as I said the

30:02

wall is split into two and

30:05

you see the red here

30:07

is the bleeding and then

30:09

you see this line of

30:11

blue dots. These are inflammatory

30:13

cells and on the

30:15

right side we have a higher

30:18

magnification and here you can

30:20

see that actually on the

30:23

left side you see the inner wall

30:26

of the aorta and then on

30:28

the right side the bleeding and

30:31

in the interface there is

30:33

this infiltration of mostly

30:35

lymphocytic cells, some macrophages,

30:37

all so there. Why

30:39

do we do this?

30:42

I mean the

30:44

dissection of the aorta you can see by

30:47

without the microscope but of

30:50

course a dissection of the aorta may

30:52

have different causes

30:55

and one is an

30:58

older person the arterial sclerosis but

31:00

if you look here on

31:03

the left side again you don't see any any

31:05

sign of arterial sclerosis here. So you

31:08

were able to rule that out as the cause? I

31:11

can rule this out. The second thing that

31:13

has to be ruled out is the genetic

31:16

defect of the connective

31:20

tissue which may lead to

31:22

this type of aortic rupture.

31:25

It appears in younger persons

31:27

and this usually

31:31

does not go along with any significant

31:34

inflammatory infiltrates. So the

31:36

inflammatory infiltrates prove in

31:38

this case that it

31:41

is not a genetic defect and

31:43

we can further make this

31:46

plausible because we did the

31:49

immunohistochemistry for the spike protein

31:51

and it is selectively located

31:54

in these areas. So

31:56

this is an additional proof. To

32:00

tie it all together, I want to

32:02

make note that this methodology

32:04

that you use was not used

32:07

during the initial autopsies. Is that

32:09

correct? Yes, this is correct.

32:12

Okay. Before the

32:14

rollout or implementation

32:16

of the COVID-19 vaccines, how

32:18

common was an aortic dissection?

32:21

As I said, we did

32:24

about 1,500 to 2,000 autopsies a year. And

32:31

I might say it might have been one

32:33

or two a year at that

32:36

time. And in this series

32:38

of 75 autopsies

32:41

that we have reexamined, we

32:44

saw five ruptures

32:48

of the aorta with

32:50

consecutive death. And

32:53

actually, in those cases where

32:55

histology from the aorta was

32:58

taken, smaller areas

33:00

of dissection, especially loss of the aelastic

33:02

fibers, I think we come to this

33:04

later, can be

33:07

proven in many, many cases, in

33:09

almost all cases. Some of these

33:11

findings are minimal. Okay.

33:15

So we know that the COVID-19

33:18

vaccines induce blood clotting. And this

33:20

was predicted by several doctors and

33:23

scientists before the COVID-19 vaccines were

33:26

made available to the general public. And

33:29

these two images here, which are not

33:31

from your own studies, but from a

33:34

general archive or published studies, show vasculitis,

33:36

which is inflammation of the blood vessels,

33:38

and that can induce blood clotting. So

33:41

can you just explain for us what

33:44

is being observed in these two images,

33:46

and in particular, what staining method was

33:48

used? When you describe

33:50

this here, it's going to help provide a

33:52

better context for the listeners as we begin

33:54

to discuss images from your own studies. On

33:57

the left side, you see the

33:59

standard A-1. and E-stain and

34:01

you see this circular structure

34:03

which is a little bit

34:06

more red. That's

34:08

the inside of the vessel

34:10

and it's thrombus formation and

34:13

you can see where the arrow

34:15

is. You can see that

34:17

the endothelium is destroyed. So one of our

34:21

main results we

34:23

stated that the endothelial damage is

34:26

one of the main causes of

34:28

the complications. What happens

34:30

if you have endothelial damage?

34:33

Well if the endothelium

34:35

is damaged and discarded into

34:37

the vessel lumen then

34:39

the basement membrane

34:42

and the extracellular matrix

34:44

of the vessel wall is exposed

34:47

to the blood and

34:49

as soon as this happens the

34:52

body wants to heal this

34:54

and the thrombocytes come and

34:56

the thrombocytes are those that initiate

34:58

a thrombus formation. So

35:00

thrombus formation actually is a

35:03

normal healing process and in

35:05

this case healing process of

35:08

damage that has been triggered

35:10

by the vaccine. Okay and then

35:13

if you would like

35:15

to go ahead and describe for us

35:17

the image on the right and I

35:19

believe that immunohistochemistry was

35:22

used in this image because we see different

35:24

colors we see some brown some black even

35:27

if you could just describe what is being

35:29

observed here. Well you see

35:31

these brown staining and

35:34

this is a fibrin so

35:36

besides thrombocytes fibrin is a

35:39

main component of thrombus

35:42

and so we can actually prove

35:45

that in this case as a

35:47

thrombus formation. And

35:49

that means a blood clot? That's

35:51

a blood clot yeah. Okay continuing

35:53

with our discussion on the blood

35:56

vessels there has been evidence of

35:58

lymphocytic inflammation of the small

36:00

blood vessels. Here we have an

36:02

image from Dr. Michael Murr. Can

36:05

you explain to us the differences we see

36:07

here from the image on the

36:09

left, which is a normal small blood vessel

36:11

compared to the image on the right, which

36:14

is a blood vessel attacked by the lymphocytes.

36:16

What observed differences do you see? Well,

36:19

actually on the left side

36:21

you see this small vessel

36:23

and inside there are these

36:25

red dots which are red

36:28

blood cells and then you can see

36:30

these elongated spindle

36:33

shaped nuclei that form

36:35

something like a wallpaper

36:39

outlining and protecting the

36:41

vessel. If

36:45

these are destroyed, as I

36:47

said before, then thrombosis

36:49

might happen and on the right side

36:51

actually you see this micro-thrombus,

36:55

which usually

36:57

mainly contains thrombocytes and

36:59

some fibrin. And very

37:04

important you see that

37:06

instead of the normal

37:08

myofibrablast that form the

37:10

vessel wall, there's an

37:12

infiltration of inflammatory cells.

37:15

Now these next two images and cases

37:17

that we will discuss are from your

37:19

own studies in which you observed vasculitis

37:21

of the small blood vessels in the

37:23

brain. This is now the second time

37:25

that we mentioned vasculitis, so just a

37:28

reminder for everybody listening that is an

37:30

inflammation of the blood vessels. You could

37:32

again for us just describe what is

37:34

being observed in these two images here.

37:37

Yes, well actually this

37:40

is one of the most alarming

37:42

findings that we had

37:44

from the beginning on that if

37:47

you really look closely

37:49

at the brain tissue

37:51

sections, you find this

37:54

vasculitis in almost all cases.

37:56

In many cases it's a

37:58

very discrete but you have to look

38:00

for it. These are

38:02

two images where you can see you really

38:05

have to look closely to see that these

38:09

small vessels in the brain, the

38:13

endothelium is swollen but then

38:15

there are these small blue

38:19

dots. These again are lymphocytes which

38:22

aggregate around these

38:25

small vessels. And lymphocytes

38:27

cause inflammation. The fact

38:30

that lymphocytes are found there means

38:32

that there is some

38:35

inflammation probably triggered by

38:38

some antigenic structure, in

38:40

this case maybe a

38:42

spike protein or something

38:45

from the vaccination. And

38:48

as I said, this is a finding

38:52

that a minimal degree

38:54

is found in almost all

38:56

of these people who died

38:58

after vaccination. And actually

39:00

we have seen it also in

39:03

one needle biopsy from

39:05

the brain. We come to this later.

39:08

And in many of

39:10

these cases which have more

39:13

pronounced inflammation of the vessels

39:16

of the brain, there

39:18

have been transient defects

39:21

like loss of speech for a

39:24

few hours, unconsciousness

39:26

for some hours, blindness

39:28

for some hours. The

39:32

brain is, if there is

39:34

no major inflammation

39:36

and no hemorrhage,

39:41

the brain is able to compensate again.

39:44

But of course this is a

39:48

very striking side

39:50

effect. commonly

40:00

seen in people who have

40:03

died post-vaccination. Yes. And

40:05

it can oftentimes individuals

40:07

with this complication have

40:09

had periods of blindness,

40:11

inability to speak properly.

40:13

Is that correct? Yes. Now

40:18

just to get this clear,

40:20

I mean they did not die from this.

40:23

This is something we find. We

40:28

find other cases where there's blood bleeding

40:31

and hemorrhage in the brain and they died

40:33

of it. But this is just a side

40:35

effect which may be compensated

40:37

and healed to a certain degree.

40:40

So somebody who is listening

40:42

right now or anybody could have received

40:45

the COVID-19 vaccine, they

40:47

could have this very issue, experience

40:49

some symptoms and not even know

40:51

that they are experiencing this? Yes,

40:54

exactly. And actually

40:57

in some cases change

41:00

in the character of

41:02

these vaccinated is reported.

41:04

Sometimes it's reversible and

41:07

apparently in some cases not. And

41:09

this may be one of the reasons. In

41:13

addition to lymphocytic inflammation, Professor

41:15

Burkhart also found other forms

41:18

of damage to small blood

41:20

vessels. These three images show

41:22

lesions of these small blood vessels in

41:24

the brain and heart. Images

41:26

A and C show these small vessels

41:29

from the brain of an 87-year-old woman

41:31

who died 302 days

41:33

after receiving a second Pfizer vaccine.

41:36

Image B shows these small vessels

41:38

from the heart muscle tissue of

41:40

an 81-year-old woman who died 23

41:43

days after receiving one dose of

41:45

the Pfizer vaccine. So

41:48

if you could just go ahead and describe

41:50

what we are seeing in these three images.

41:53

Well in all three images you see

41:55

lesions of the blood vessels, of the

41:57

smaller blood vessels. And

42:00

on the left side, this is a

42:03

small vessel from the brain and

42:05

in this case, there is no

42:09

major inflammation but we

42:11

have this blue stain and the

42:14

blue stain means iron deposition. And

42:16

iron is deposited where there has

42:19

been bleeding before.

42:21

So the erythrocytes contain iron

42:23

and it is deposited

42:26

in the tissue

42:29

as what we call

42:31

hemosidirin. And hemosidirin is

42:33

a very strong indication of

42:37

bleeding in

42:39

this vessel ball. Now this person at

42:42

this point at least was lucky

42:45

because this bleeding was

42:47

stopped within the vessel ball and

42:49

it did not go outside into the

42:52

brain tissue. And if you look

42:54

at the right side, again this is from

42:56

the brain and this is

42:58

a stain which we call the

43:01

Congorette but it stains especially the

43:03

elastic fibers and usually

43:05

these small vessels

43:08

are surrounded, completely surrounded

43:10

by elastic fibers so

43:13

that they will

43:15

not rupture. And you

43:17

can see that in this

43:19

part, there are elastic fibers.

43:22

Now they are also not

43:24

normal. They are clumped together

43:26

and discontinuous but in this

43:28

part, they are completely lacking

43:31

and you see that the small

43:34

vessel has what

43:36

we call an aneurysm. And now

43:38

this of course could rupture at any

43:40

time because there is no elastic

43:44

lamellar which

43:46

may contain it. And in the

43:48

middle, you see that also

43:51

the small vessels in the heart

43:53

muscle are affected and in this

43:55

case, the endothelium

43:58

is swollen. and

44:00

there has been a deposition

44:02

of some

44:04

acellular red stain

44:06

material which is apparently

44:09

related to what we call amyloid

44:11

and in some cases also related

44:14

to prions.

44:17

And these are proteins that

44:20

may be derived from the

44:22

spike protein and the deleterious

44:26

effect is that the body cannot

44:28

get rid of them. They

44:30

are not digestible by

44:33

macrophages or other inflammatory

44:35

cells. The image in the

44:37

middle, it is labeled amyloid protein deposits.

44:39

Can you just explain what that is?

44:42

Well, this is a

44:44

strange type of protein.

44:48

It is a misfolded protein

44:51

which by this unnatural

44:54

structure cannot be disintegrated

44:56

by the body. And

44:59

so it remains in the body. And

45:03

as I said, there is a

45:05

certain disease, amyloidosis, which

45:09

is very rare and develops after

45:11

many years of infections and something

45:14

like that. But

45:16

we find similar

45:18

proteinaceous deposits after

45:21

exclamation and they

45:25

are probably not

45:27

identical, but they are related to

45:29

it. That is why we call

45:31

it amyloid-like. Now how

45:34

do these changes to the blood vessels affect

45:36

organ function? Well, if you look

45:38

at the middle picture, of course you can see

45:40

that this small vessel is practically

45:43

occluded. I mean there is

45:45

maybe one third only open.

45:47

So of course if

45:49

you have some trouble

45:51

with perfusion of the myocardium,

45:55

these small vessels are occluded in

45:57

many areas. you

46:01

might actually die of a heart failure,

46:04

but this is not a heart

46:06

failure by arterial sclerosis or something

46:08

like that, but it's in

46:12

the larger sense, what we call

46:14

a small vessel disease. Small

46:17

vessel disease is also detected

46:21

in some cases of intoxication, this

46:23

other materials, and in this case,

46:25

apparently it's a spike protein. In

46:29

this next case, Professor Burkhart observed

46:31

a saborachnoid hemorrhage in a 29

46:33

year old male who

46:35

received one dose of AstraZeneca and

46:38

one dose of Cesar. He

46:40

died 46 days after the

46:42

second injection. Most cases

46:45

of saborachnoid hemorrhage arise from

46:47

structural defects of brain vessels,

46:50

most often aneurysms. However,

46:52

no such defects were found in

46:55

this case, nor did the patient

46:57

have any other known illnesses. First

47:00

of all, before we discuss the

47:02

image from your study, can you

47:05

just explain briefly what a saborachnoid

47:07

hemorrhage is? Well,

47:09

the brain is covered by a

47:11

very delicate kind of skin, and

47:19

inside there are small vessels which

47:22

also supply the brain. And in

47:24

this image, actually, this is the

47:26

surface of the brain. You see

47:28

the surface of the brain here,

47:31

and there's this very delicate structure

47:34

which is the saborachnoid

47:36

membrane with a small

47:38

vessel here. And then

47:40

we have the brain groove

47:42

here, and here

47:45

a little bit larger vessel is shown,

47:47

and you can see that the vessel

47:50

wall, if you would look at it

47:52

at a high magnification, is dissolved,

47:54

and you can see that there's blood

47:57

also in the surrounding.

48:00

of this vessel, so this vessel apparently

48:03

is not containing

48:05

the blood anymore, but there's a

48:07

hemorrhage. And in this patient,

48:10

no larger aneurysm was

48:13

found because this

48:15

type of zapachorrhizae,

48:19

bleeding usually occurs or

48:22

may occur in younger persons,

48:24

but it is caused by

48:26

genetic defect in the larger

48:28

vessels of the brain basis.

48:31

And this was not the case in this

48:33

patient. It was not found. So he had

48:36

to diffuse hemorrhage

48:38

from inflamed and

48:40

partly destroyed smaller vessels.

48:44

Do you know what symptoms that he may have

48:46

had and if not, what symptoms

48:49

would one experience if

48:52

they were suffering with this particular condition?

48:55

Would they have any symptoms? Well,

48:57

actually this was one of these cases.

49:01

He was suddenly unconscious and

49:04

reanimation was done in the

49:06

hospital, but he died. Actually

49:09

they could not. Did he die

49:12

suddenly? Yes, actually. Excuse me. Sure.

49:16

Before he was unconscious, he had

49:18

convulsions. Okay. And

49:22

after that, he was

49:24

resuscitated and died in

49:26

the hospital and then came back to

49:28

the hospital. At age

49:30

29, otherwise healthy male? Yes,

49:33

yes. Okay. Now

49:36

what persuaded you to conclude that it

49:38

was possible that it was the COVID-19

49:41

vaccine that

49:44

was perhaps associated with this death,

49:47

this ailment? Well

49:51

actually, in addition to

49:53

these lesions in the brain, we

49:55

found myocarditis

49:57

also, which would

50:00

be very unusual that you have

50:02

brain hemorrhoids

50:04

and myocarditis and

50:07

we had endothelial lesions, damage

50:10

and destruction of endothelium, especially

50:12

not only in the brain

50:14

as I showed you but

50:16

also in the muocardium. So

50:18

this is a apparently

50:21

toxic effect and

50:25

the assumption that this is

50:27

a toxic effect mediated by

50:29

the spike protein is made

50:31

further very

50:33

probable because we could show the spike

50:36

protein in these lesions and by the

50:38

way this is the person where we

50:40

found the spike protein in the

50:43

testis in the spermatogenic

50:46

cells. Yes,

50:49

and we will get to that but I just want

50:51

to speak generally here. You

50:53

are able to find several abnormalities

50:55

in one patient. Yes. What

50:58

does that suggest to you? Well,

51:01

that would suggest that he did

51:03

not die of a brain aneurysm

51:05

but he died of multiple

51:10

lesions which probably are caused

51:12

by the same in this

51:14

case toxic agent. Thank

51:17

you. I now want to

51:19

have you take a look here at some

51:21

of the findings from German-Swedish pathologist Jute Krueger.

51:24

I think it's important to highlight that there

51:26

are other pathologists as well who are doing

51:28

this work who are seeing the same abnormalities.

51:30

So I'd just like you to provide a

51:33

comment on what's being shown here and its

51:35

implications. Well actually

51:37

this is exactly what we see too.

51:40

On the left there's a normal

51:43

artery. I mean it's

51:45

H and E stain so

51:47

you don't see the elastic fibers. If

51:49

you would have an elastic stain you

51:51

would see some defects there and

51:54

here you can see the inflammatory

51:56

infiltration in the intimal part

51:58

of the vessel. and

52:01

there's no endothelium here to be

52:03

seen. And

52:06

if there's no endothelium, then thrombus

52:08

is formed. How

52:11

are you able to determine if

52:13

these lesions, which are tissue abnormalities,

52:16

are a result of the

52:18

COVID-19 vaccine or the COVID

52:20

virus? The

52:22

pathogenic agent is

52:25

in both cases identical,

52:30

especially if a spike protein, of

52:33

course, is induced by

52:35

the vaccination and

52:37

the spike protein is also produced by

52:39

the virus. So the

52:42

levels of the spike protein is

52:45

very much lower in viral

52:47

infected persons in contradistinction to

52:49

those who have been vaccinated.

52:52

And actually, nobody knows

52:54

how high these concentrations

52:56

of the spike protein can

52:58

be in the

53:01

vaccinated. And of course, we know it

53:04

can persist for many months now and

53:07

can be found in all organs, while

53:09

in the normal infection, it usually

53:12

stays limited to the aerodigestic

53:16

tract. And in

53:19

addition to the spike protein,

53:21

of course, the true viral

53:24

infection has other antigenic structures.

53:27

And one of these other

53:30

antigenic structures is the

53:32

nucleocapside. And

53:34

so if we find

53:37

the spike protein and the nucleocapside, then

53:40

it is probably the result of

53:43

a true viral infection. But if

53:45

we find only spike protein and

53:48

no nucleocapside, this

53:51

is a very strong suggestion

53:53

that this is a consequence of

53:56

the vaccination. we

54:00

know that the spike protein expression is

54:02

caused by the vaccination and not the

54:05

virus. To best

54:07

understand this, it is important

54:09

to note that SARS-CoV-2 virus

54:11

particles contain two major proteins.

54:14

First, the spike protein, which is

54:16

located at the surface of the

54:18

virus particle. And second,

54:20

the nucleocapsid protein, which forms

54:23

a protective layer around the

54:25

RNA genome. Therefore,

54:27

virus-infected cells should make

54:29

both of these proteins. Meanwhile,

54:32

the vaccine only encodes the

54:35

spike protein and not the

54:37

nucleocapsid protein. This

54:39

has been experimentally confirmed by

54:41

German physician Dr. Michael Mertz.

54:45

How can we determine whether the

54:47

spike protein and or the nucleocapsid

54:49

protein had been present in the

54:51

patient's tissues? We can

54:54

use immunohistochemistry or

54:56

IHC. This method

54:58

allows for the detection of specific

55:00

molecules of interest in the tissue

55:03

samples by using specific antibodies. In

55:05

this case, the molecules of

55:08

interest are the spike protein

55:10

or the nucleocapsid respectively. We

55:13

will hear you see spike protein as

55:15

the example. Let's now

55:17

summarize this technique. First,

55:20

an antibody which interacts with the

55:22

molecule of interest, in this case

55:24

the spike protein, is applied to

55:27

the tissue sample. After

55:29

allowing some time for the antibody

55:31

to bind to its target, the

55:33

unbound surplus is washed off. Next,

55:37

a secondary antibody coupled with

55:39

the catalytic protein enzyme is

55:42

applied to the same tissue sample. This

55:44

second antibody binds to the first

55:47

one. After some more

55:49

time, the unbound surplus is again

55:51

washed off. Third, a

55:54

colorless dye precursor, most often

55:57

diaminobenzidine, is added to the

56:00

This dye precursor can be converted

56:03

to an actual dye by the

56:05

enzyme that is attached to the

56:07

second antibody, and nuts indirectly to

56:09

the spike protein. The brown

56:11

dye that is produced by the enzyme

56:13

is insoluble, so it comes out

56:16

of solution and is deposited close

56:18

by. However, there

56:20

are deposits of brown pigment, we

56:23

know that spike protein must have

56:25

been present. We can't

56:27

perform these same procedures separately with

56:29

an antibody that specifically recognizes

56:32

the nucleocapsid. And

56:34

now we're going to focus

56:36

on the use of immunohistochemistry

56:38

to detect vaccine-induced expression of

56:40

the spike protein. Here

56:43

we have two images, and

56:45

these images come from Dr. Michael Mertz.

56:48

And they show the cross-sections through

56:50

two small blood vessels. Can you

56:52

explain what is being observed here?

56:55

Yes, I think these pictures illustrate

56:57

exactly what I just tried to

56:59

explain. I mean, you see on

57:02

the top, you see small

57:04

vessels. And

57:08

already from this magnification,

57:10

you can see that

57:12

these vessels have an

57:14

endothelial damage. Then you have the

57:17

brown stain, and that means that

57:20

immunohistochemistry spike protein can

57:22

be seen here. Below,

57:25

the same vessels are

57:27

seen, and they are stained for

57:29

nucleocapsid. And you see

57:31

the same lesions, of course,

57:34

of the endothelium, but there's no staining. So

57:36

this is a very strong indication.

57:39

In considering everything that

57:41

you see, that this is

57:43

a specific lesion by

57:46

the vaccine produced

57:48

as spike protein, and not by

57:50

the virus produced as spike protein.

57:54

In each of the cases, did

57:56

you perform nucleocapsid control? In

57:58

all the cases, did you perform nucleocapsid control? that

58:00

we have a positive spike

58:03

protein reaction, we do the

58:05

nucleocapside with negative at positive

58:08

controls. And if

58:10

the spike protein is negative of course, then

58:13

we don't have to do

58:15

the nucleocapside because this

58:17

is not relevant

58:20

anymore. And actually we have

58:23

I think two or three cases where we

58:26

do have expression of

58:28

a nucleocapside. So in

58:32

these cases of course, there might be

58:35

an additive effect of the

58:37

vaccination and a viral infection

58:39

either before or after vaccination.

58:42

And in our observation these are

58:45

the cases with the most severe

58:47

side effects. When

58:49

somebody has been infected and

58:51

then also was vaccinated, you

58:54

see the most severe symptom.

58:56

We cannot prove this scientifically

58:59

by now, but this is

59:01

just a case observation.

59:04

Thank you. That is a very important

59:07

point that you've raised here. While

59:11

detection of spike protein clearly points

59:13

to vaccine causation, Professor Burkhardt does

59:16

not rely on this method alone.

59:18

Let me

59:21

stress this. We never

59:23

make this diagnosis just solely

59:26

only dependent on our

59:28

immunohistochemistry. We

59:31

only make this diagnosis if

59:33

we have lesions which

59:35

are distinctly positive and which

59:38

by themselves are already

59:40

more or less absolutely

59:44

typical of vaccination damage

59:46

like elastic

59:48

reconstruction. This

59:52

next image shows the expression of

59:54

spike protein in the coronary artery of a

59:56

24 year old male with no

59:59

known prior illness. The

1:00:01

young man received one dose of Johnson

1:00:03

& Johnson and one dose of Hyther.

1:00:06

He died 56 days after the

1:00:08

second injection. Can

1:00:11

you just describe the significance of

1:00:13

this image? Well, you see

1:00:15

the vessel wall on the

1:00:17

left lower corner and then

1:00:19

you see the split in the middle. And

1:00:22

here you can see a dense infiltration

1:00:25

of lymphocytes. These are the

1:00:27

small dark dots and

1:00:30

then you can hear this is

1:00:32

a thrombus and we did

1:00:34

the spike protein and it is

1:00:36

positive in some of these inflammatory

1:00:38

cells and maybe we

1:00:40

are not very sure what it

1:00:43

means in the thrombus but this

1:00:46

might be an artifact. And this

1:00:48

by the way is an artifact too. But

1:00:51

given that this image shows a lot

1:00:53

of brown pigmentation, does that mean that

1:00:55

there's a lot of spike protein being

1:00:57

observed here? Yes,

1:00:59

yes. I mean here

1:01:03

in this line should have

1:01:05

been the endothelium protecting the

1:01:07

blood which has been flown

1:01:10

here and this is the

1:01:12

wall. And here

1:01:14

the endothelium should have made

1:01:16

a borderline between the

1:01:18

two and it is

1:01:21

destroyed. This inflammatory infiltration

1:01:23

and in this

1:01:25

context also some thrombocytes have

1:01:27

been attached and then the

1:01:30

process of thrombotic event was

1:01:32

started. What

1:01:34

role did the spike protein have in the

1:01:37

formation of the blood clot here? Well,

1:01:41

it's the endothelial damage. It's a

1:01:43

destruction of the endothelium whether

1:01:46

it's by toxic or by immunologic

1:01:49

interaction. It's

1:01:53

not clear in every case but in any

1:01:55

case there's

1:01:57

no endothelium here. Just

1:02:00

like that. It

1:02:03

is now well established that there

1:02:05

is an increased risk of

1:02:07

myocarditis after vaccination. Professor Burkhart

1:02:09

further established in this particular

1:02:11

case that the severe myocarditis

1:02:14

observed in the patient was

1:02:16

by protein induced. The

1:02:18

patient was a 54 year old

1:02:20

woman who died 11 days

1:02:22

after receiving a second Pfizer

1:02:24

injection. I'll

1:02:26

let you just describe here what we

1:02:28

are able to see. Well

1:02:30

on the left side of course you

1:02:33

see a hard muscle with very

1:02:35

pronounced disintegration and destruction

1:02:38

of muscle cells. And there

1:02:40

is a dense infiltration again

1:02:42

of these small dots that

1:02:44

are the lymphocytes. And in

1:02:48

contrast to infarction

1:02:51

there are no neutrophiles,

1:02:54

granulocytes, which are the

1:02:56

cells that are predominant in a

1:02:59

myocardial infarction. And

1:03:01

on the right side it is just

1:03:03

shown that the spike protein is

1:03:06

found in these destroyed muscle

1:03:09

cells. In this

1:03:12

case the autopsy done

1:03:14

by the pathologist was

1:03:16

death by cardiomycontinuation, which

1:03:18

of course contains

1:03:20

everything. I mean it

1:03:23

is not an

1:03:26

etiological diagnosis, it's

1:03:28

just a statement

1:03:31

of a

1:03:33

plausible cause of death. Another

1:03:37

pathologist, Dr. Michael Mertz, is

1:03:40

also using immunohistochemical staining to

1:03:42

detect spike protein in the

1:03:44

tissues of vaccinated persons. These

1:03:47

two images are from Dr.

1:03:49

Mertz published study in which

1:03:51

he examined the brain tissue

1:03:54

of a vaccinated patient who

1:03:56

developed myocarditis and encephalitis post-injection.

1:03:59

The patient was a... 76-year-old male

1:04:01

who had received one dose

1:04:03

of AstraZeneca and two doses

1:04:05

of Pfizer. He died

1:04:07

three weeks after the third injection.

1:04:11

Would you like to just comment on what is

1:04:13

being observed here? On the left we see this

1:04:15

spike and then on the right we see the

1:04:17

nucleocapsid control. Well,

1:04:19

yes, you see the brain

1:04:21

tissue and the larger

1:04:24

cellular elements are the

1:04:26

nerve cells and on the left side

1:04:28

you see a positive staining,

1:04:31

in this case a brown

1:04:33

staining of some of these

1:04:36

neural elements and on the right

1:04:38

side again a negative finding. So

1:04:40

this is a strong indication that

1:04:43

these damages are caused by

1:04:45

this vaccine-induced

1:04:47

spike protein. So

1:04:49

just to reiterate one more time

1:04:51

for the listeners, the nucleocapsid control

1:04:53

which is being shown here on

1:04:55

the right indicates

1:04:58

in this case that the

1:05:00

expression of the spike protein

1:05:02

was caused by the COVID-19

1:05:04

vaccination and not the

1:05:06

COVID-19 virus. Yes,

1:05:09

this in the context of the whole...

1:05:11

In the context again... Of

1:05:13

all findings and all clinical data,

1:05:16

yes. Yes, thank you. Okay,

1:05:19

now we can have a discussion about

1:05:22

encephalitis and I want to talk about

1:05:24

a case in which a brain biopsy

1:05:26

was done and to be honest I

1:05:28

was very surprised by this

1:05:30

particular case because I did not

1:05:32

know that one could have a

1:05:34

brain biopsy. From my understanding it's

1:05:36

done very rarely. So

1:05:38

can you talk about why in this case

1:05:40

a brain biopsy was done on this particular

1:05:42

individual? Well actually

1:05:45

a needle biopsy of

1:05:48

brain lesions actually is

1:05:50

something that it

1:05:53

must be very rare because in 40 years I have

1:05:55

not had a needle... biopsy

1:06:00

of the brain. What I

1:06:02

did have is fast-frozen

1:06:04

sections to open up a

1:06:08

surgery of the brain of course. If

1:06:10

they open the brain

1:06:12

and find a tumor or something

1:06:14

and then they make a fast-frozen

1:06:18

section to get the diagnosis

1:06:20

during a surgery. But

1:06:23

in this case she had severe

1:06:27

neurological symptoms. Of

1:06:30

course they did investigate the

1:06:32

spinal fluid and everything but they did

1:06:35

not come to a conclusion. So there

1:06:38

was a suggestion of a tumor in

1:06:40

the brain and they

1:06:43

suspected the malignant lymphoma. And

1:06:46

a malignant lymphoma can be very

1:06:48

successfully treated if you know

1:06:50

the exact type. So

1:06:53

they were desperate to find out

1:06:55

what type of a lymphoma was

1:06:57

here to give her the

1:06:59

right treatment. But then

1:07:02

they did the needle

1:07:04

biopsy and no

1:07:08

tumor, no cancer

1:07:10

was found. This has

1:07:12

been confirmed by several university pathologists

1:07:18

who looked at it. But instead

1:07:20

they found a very

1:07:22

pronounced vasculitis

1:07:25

and also a

1:07:28

concomitant encephalitis. On

1:07:30

the left side you see this

1:07:33

is the needle, the contents

1:07:35

of the needle that was used to get the

1:07:38

tissue out of the brain. And on

1:07:40

the right side you see a high

1:07:42

magnification and also in this overview

1:07:45

you can see these spots

1:07:47

where there is a more

1:07:49

dense aggregation of cells. These

1:07:51

are inflammatory cells and they

1:07:53

are located selectively

1:07:55

around small vessels.

1:07:58

So this is definitely a good example. definitely

1:08:00

a vasculitis, a

1:08:03

lymphocytic vasculitis and

1:08:05

also some inflammation in

1:08:07

the surrounding tissue could

1:08:09

be found. So there

1:08:11

was also a codcomitant

1:08:13

encephalitis. Melic syndrome

1:08:16

form was excluded and

1:08:18

vasculitis and encephalitis was

1:08:20

confirmed and I suppose

1:08:23

she is treated now

1:08:25

probably with

1:08:27

some anti-inflammatory agents.

1:08:31

Professor Burkhart also tested this

1:08:34

same brain biopsy sample for

1:08:36

the spike protein using immunohistochemistry.

1:08:39

On the left side you see the

1:08:41

spike protein and it is selectively expressed

1:08:43

in the vessel walls

1:08:46

of the small vessel. It is

1:08:49

brown stained and there is not

1:08:51

much background in this case. On

1:08:54

the right side you see the

1:08:57

nucleocapside which is absolutely negative

1:08:59

in this case. In

1:09:01

the middle you can see that these large

1:09:04

nerve cells also express the spike

1:09:06

protein so this is an indication

1:09:08

of a codcomitant encephalitis.

1:09:13

Michael Mertz was also able to find in

1:09:15

his case study of a 76-year-old man. He

1:09:19

found that this individual who

1:09:21

was also vaccinated had encephalitis

1:09:24

as well. So is encephalitis

1:09:26

potentially a common

1:09:29

ailment associated with COVID-19 vaccination?

1:09:33

Well apparently yes. As

1:09:36

I said we find minor

1:09:39

lesions of the small vessels

1:09:42

and if the small vessels

1:09:44

are inflamed the

1:09:49

so-called blood-brain

1:09:51

barrier breaks down and that

1:09:53

means that the vaccine

1:09:56

contents can enter

1:09:59

into the brain. And I

1:10:01

think just recently some

1:10:04

Japanese investigators found that

1:10:06

the spike protein is

1:10:08

selectively toxic for ganglion

1:10:10

cells, for nerve cells.

1:10:13

Once it gets into the brain,

1:10:15

it may cause encephalitis.

1:10:20

This image shows the strong expression

1:10:22

of spike protein within the spleen

1:10:24

of a 94-year-old female

1:10:26

who died 67 days

1:10:28

after the second injection. Spike

1:10:31

protein expression in the spleen is

1:10:33

notable for two reasons. Firstly, we

1:10:35

know that vaccine particles tend to

1:10:38

accumulate in the spleen. Secondly,

1:10:40

it is a major lymphatic organ.

1:10:43

Spike protein expression in the spleen may

1:10:45

therefore result in the killing of many

1:10:48

lymphocytes, which would in turn

1:10:50

lead to immunosuppression. Can

1:10:54

you explain what is being observed here with this

1:10:56

case? And why does

1:10:58

the spleen show such strong expression

1:11:00

of the spike protein? Well,

1:11:04

actually, of course, this is one

1:11:06

of the aims of the vaccination.

1:11:11

They want to provoke the

1:11:13

immune system to produce a

1:11:16

spike protein. But in this case,

1:11:19

it was a very strong expression

1:11:21

and not only in the spleen.

1:11:23

I just took this picture to

1:11:25

show that the

1:11:27

vaccination does what it should do, but

1:11:30

it does too much. In

1:11:33

some cases, it's what

1:11:35

we call lymphocytic amok.

1:11:37

Yes, this is a term

1:11:39

or phrase that you have coined, really. You

1:11:42

coined the term lymphocyte amok. What do you

1:11:44

mean by that? Well, I

1:11:46

mean that the

1:11:48

lymphocytes are overstimulated.

1:11:53

I mean, the vaccination wants to

1:11:55

stimulate them, of course, but in

1:11:57

some cases, they are overstimulated. And

1:12:00

clinicians talk about hyperinflammatory

1:12:03

syndrome, and this may

1:12:05

be destructive in many organs. And

1:12:08

this always has a danger of

1:12:11

an autoimmune disease, which we talked

1:12:13

about earlier about the thyroid, glucose,

1:12:15

and so on. So

1:12:18

how serious is immunosuppression, for example?

1:12:20

And how does it typically present

1:12:22

itself in patients who may be

1:12:25

suffering from it? Infosuppression

1:12:31

is something that is not

1:12:33

conspicuous in our cases, because

1:12:35

we see overstimulation, like

1:12:37

in this picture, and sometimes

1:12:40

we see depletion of the

1:12:42

spleen and lymph nodes. But

1:12:45

of course, by

1:12:48

the morphological structures, we

1:12:51

cannot make

1:12:53

any statement about the state

1:12:55

of the immune

1:12:57

system. OK. So

1:13:00

this next case involves a

1:13:03

35-year-old woman who was vaccinated

1:13:05

and started to experience skin

1:13:07

lesions. And this has

1:13:09

had a severe impact on her life.

1:13:12

I have heard you speak about her

1:13:14

case publicly before. Can

1:13:16

you just explain for us what

1:13:18

this woman was experiencing and how this

1:13:20

impacted her quality of life? Well,

1:13:23

first of all, she had several severe

1:13:26

side effects. But

1:13:29

the one side effect that affects

1:13:31

her or reduces her quality of

1:13:34

life the most is the skin

1:13:37

lesions. She had an

1:13:39

absolutely healthy skin before, and now

1:13:41

she does not

1:13:43

want to enter into the swimming

1:13:46

pool or open.

1:13:50

And actually, her

1:13:52

whole skin is covered with these

1:13:56

pustular lesions. And

1:13:58

we took a biopsy. And you

1:14:00

can see on the left side that

1:14:02

this is something that is related

1:14:05

to what we call a lichen

1:14:08

plane of that is an

1:14:10

autoimmune disease destroying the basal

1:14:12

cells. And you can see that

1:14:15

here these cells

1:14:18

are specifically stained by the

1:14:20

spike protein and they are

1:14:22

vacuolated. That means they are

1:14:24

damaged or even dead. And

1:14:27

then you can see the spike protein

1:14:29

also in this lymphocytic infiltrate. The lymphocytic

1:14:32

infiltrate is the one that attacks the

1:14:34

basal cell. So you did find the

1:14:37

spike protein expressed in the skin

1:14:39

biopsy. So I do want to

1:14:41

ask you, could skin biopsies also

1:14:43

be useful in potential vaccine damage

1:14:45

to organs other than the skin?

1:14:47

Yes. Well first of all,

1:14:50

I said this is a lesion

1:14:52

that is related to

1:14:54

what is known as lichen

1:14:57

planus. But it

1:14:59

is an atypical type

1:15:01

because in addition to these destructions

1:15:04

of the epidermis, we

1:15:07

also find the vasculitis. And

1:15:09

this is not a typical

1:15:12

feature of this disease lichen

1:15:14

planus. So we have

1:15:16

an atypical autoimmune disease

1:15:19

with concomitant vasculitis.

1:15:23

And we get a lot of skin

1:15:26

biopsies now with the question. And

1:15:29

there are two different types of

1:15:31

questions. One are the

1:15:33

persons that have lesions of the

1:15:35

skin. And there

1:15:38

we find atypical lichen planus

1:15:40

and what is called pentaglytes

1:15:43

lesions which are autoimmune

1:15:46

diseases. They have lesions

1:15:48

of the skin. But then we have

1:15:50

other persons that do not have any

1:15:52

skin lesions but other

1:15:56

side effects. And here we

1:15:58

find this vasculitis. of the

1:16:00

skin and this is

1:16:02

very clear and it is

1:16:04

also associated with spike protein

1:16:07

expression in the endothelium. So

1:16:09

there are these two

1:16:11

possibilities. Many

1:16:14

women have experienced and

1:16:16

reported on menstrual disruptions

1:16:18

post-vaccination and you

1:16:20

have been able to see this in

1:16:22

your own studies. In particular, there was

1:16:24

one woman, a 52-year-old woman who was

1:16:26

still having a menstrual cycle and

1:16:29

post-vaccine she began to experience very

1:16:31

heavy bleeding. These three images here

1:16:34

are showing the tissue of the

1:16:36

endometrium which is the lining of

1:16:38

the uterus and I see that

1:16:41

on the right we do see

1:16:43

the nucleocapsid control which is important

1:16:45

because again that is able to

1:16:47

indicate that the expression of the

1:16:49

spike protein here is a result

1:16:52

of the COVID-19 vaccine and not the virus but

1:16:54

I will let you take it from here. What

1:16:56

are we seeing in these three images? Well,

1:16:59

first of all let me

1:17:01

add this woman

1:17:05

not only had these disastrous

1:17:07

bleeding problems but she also

1:17:09

had other very

1:17:12

severe side effects,

1:17:14

neurologically, blood perfusion

1:17:16

and so on. She is really

1:17:19

very sick but nobody

1:17:22

took her seriously so

1:17:24

this abrasio was done

1:17:26

and we can

1:17:28

see on the left side we see

1:17:30

the glands of the

1:17:32

endometrium and you can see that

1:17:35

the epithelial cells are positively

1:17:37

stained. The background, the stroma

1:17:39

what we call it is

1:17:41

negative so this is an

1:17:43

indication that this is a

1:17:45

specific stain and also in

1:17:47

this case the nucleocapsid

1:17:50

is negative.

1:17:53

Now what you see here these

1:17:55

are red blood cells,

1:17:57

these are bethrids so this is not a

1:18:00

immunohistochemical staining. But

1:18:03

what is very striking, and I have

1:18:05

never seen this before, and I looked

1:18:07

into textbooks about

1:18:09

the formation of

1:18:12

lymphopolycurls, small

1:18:14

lymph nodes, so to say, in

1:18:17

the endometrium. And there are

1:18:20

some references to lymphoplasmocytic

1:18:24

endometritis, but I

1:18:27

didn't find the term lymphonoma nodular

1:18:31

endometritis. And actually we find, I

1:18:34

think even non-pathologists may

1:18:36

see that this is a small

1:18:39

nodule here of dense,

1:18:41

aggregated cells. These

1:18:43

are lymphocytes. And

1:18:45

this you would call a lympholica.

1:18:48

And in the middle here, you

1:18:50

can see these stains, and there's a gland.

1:18:53

And this gland expresses a

1:18:56

spike protein. So actually

1:18:58

we have the autoimmune

1:19:01

attack inflagranti here

1:19:05

in the endometrium. Great

1:19:07

explanation there. So this, what we're seeing

1:19:10

in the middle image, you say is

1:19:12

highly unusual, what you just described. Okay.

1:19:15

And the excess bleeding

1:19:17

that this woman had experienced, that is

1:19:19

attributable to the COVID-19 vaccine? Well,

1:19:24

I think this is a very

1:19:26

strong case. I mean, by

1:19:28

what she had, of course

1:19:31

it could be menopausal bleeding,

1:19:33

but as I said, she

1:19:35

has other very strong symptoms,

1:19:38

side effects. Lymphocytic

1:19:42

inflammation and spike expression is

1:19:44

also observed in the testis.

1:19:47

The image on the left stained with H.E.

1:19:49

is from a 55 year old male who

1:19:51

died seven days after receiving

1:19:53

a second Pfizer injection. The

1:19:56

image on the right shows spike protein

1:19:58

expression in the sceminar. which

1:20:01

are the cells that produce ferns. This

1:20:03

sample is from a 29-year-old male who

1:20:06

died 46 days after

1:20:08

his second injection. As

1:20:10

discussed earlier, the immediate cause of

1:20:12

his death was a subarachnoid hemorrhage.

1:20:16

Well, first of all, on the

1:20:18

left side you see a distinct

1:20:21

lymphocytic nodule forming around the blood

1:20:23

vessel. This

1:20:26

is lymphocytic pesculitis and

1:20:28

this is a section from

1:20:30

the test test. On

1:20:32

the right side you can

1:20:35

see the spermatogenic tubules of

1:20:37

the test test. You

1:20:39

can see that the

1:20:41

stratification usually is very

1:20:44

regular and you can see it is

1:20:46

disturbed. Usually

1:20:48

in the middle, in the

1:20:50

lumen, there should be lots

1:20:53

of spermatocytes. You

1:20:55

can see a few spermatocytes here.

1:20:58

These are the very small elements.

1:21:01

But what

1:21:03

is conspicuous is that these

1:21:05

larger elements, which are the

1:21:08

spermatogonia, are the cells

1:21:10

that form the sperms,

1:21:13

are detached in the

1:21:15

lumen of these small

1:21:18

canals. The image on

1:21:20

the right side, labeled

1:21:22

spike protein, do

1:21:24

you see sperm cells in this

1:21:26

image here? If

1:21:29

you look exactly, you can find

1:21:31

one or two in one of these

1:21:34

canals, but usually there should be

1:21:36

at least 20 or 30 or even more. Usually

1:21:41

it's filled. And

1:21:44

especially the stratification is

1:21:46

completely destroyed. Would

1:21:49

you expect someone to have symptoms if

1:21:51

they were to be experiencing this

1:21:54

complication? Well,

1:21:56

I wouldn't think

1:21:58

that they have any symptoms. any dramatic

1:22:03

symptoms but probably

1:22:06

sexual activity

1:22:08

would be lowered because it

1:22:10

is connected with the spermatocyte

1:22:13

production. And by the

1:22:15

way, we also found expression

1:22:18

of spike protein in the prostate gland.

1:22:20

So this is also part of the

1:22:23

segment. So

1:22:25

it should be effective but probably not

1:22:29

everybody would take

1:22:31

a record of it. During

1:22:35

a recent speech in Stockholm, you

1:22:37

said that you would

1:22:39

recommend a woman of childbearing age

1:22:41

to not become pregnant by a

1:22:43

man who has been vaccinated. Can

1:22:46

you expand further on what you meant by that?

1:22:49

Well, actually, I

1:22:51

wanted to add something but I was interrupted

1:22:54

by a big

1:22:57

applause. If

1:22:59

I may make a personal comment,

1:23:02

this is not a scientific comment. If

1:23:07

I were a woman in a fertile

1:23:09

age, I would not plan

1:23:12

a motherhood from a person, from

1:23:14

a man who has been vaccinated.

1:23:24

I think these pictures are very disturbing

1:23:26

for me. And

1:23:29

I said, unless and then I

1:23:32

stopped and I get

1:23:34

many telephone calls of

1:23:36

women who say, well, what did

1:23:38

you say, unless what? Yeah.

1:23:41

So right now, why don't you tell us, what else are you going to say

1:23:43

at that moment? I would at least

1:23:47

wait for two

1:23:50

or three cycles of spermatogenesis.

1:23:52

Now the cycle of spermatogenesis

1:23:54

is about 70 days. So

1:23:58

I would wait for... let's

1:24:03

say,

1:24:07

three quarters of a year or something like that.

1:24:10

And before this I

1:24:12

would suggest to make

1:24:14

a spermatogram, examine

1:24:18

this sperma and

1:24:20

especially the totality

1:24:23

of the sperma.

1:24:26

So I think this would be an

1:24:29

indication. Now as far as I

1:24:32

have seen and I

1:24:34

have not only seen the testes

1:24:37

of this young man but also

1:24:39

of older men but of course

1:24:41

they are more difficult

1:24:43

to interpret. But

1:24:46

as far as I can see

1:24:48

the spermatocytes themselves do

1:24:50

not express the spike

1:24:53

protein as far as I

1:24:55

know by now. Anyhow this is of course

1:24:58

an alarming finding. Sure

1:25:02

and according to data submitted by

1:25:04

Pfizer to the Japanese health regulator

1:25:07

the vaccine particles do distribute

1:25:10

to the ovaries but

1:25:13

in your own studies were you able

1:25:15

to find spike protein expression in the

1:25:17

ovaries? Yes, we

1:25:21

did find this. Unfortunately these

1:25:23

are mostly elderly women where

1:25:26

we get specimens from the

1:25:28

ovary. Unfortunately

1:25:32

during autopsy the ovaries

1:25:34

are not taken

1:25:36

for histological

1:25:39

examination. So

1:25:41

this has to be

1:25:43

interpreted very cautiously. We

1:25:46

find it actually mostly in the

1:25:49

vessels in the vessel walls

1:25:51

of the ovary. But

1:25:55

we are behind this question. Professor

1:25:58

Burkhart a few times throughout this interview overview, we

1:26:00

did mention the elastic fiber. So that is

1:26:02

now what I would like to focus on.

1:26:05

And you have been able to show

1:26:07

that there is damage to the elastic

1:26:09

fibers caused by the COVID-19 vaccines. But

1:26:11

before we discuss some of the images,

1:26:13

I would like you to describe in

1:26:16

simple terms, the function of elastic fibers

1:26:18

in the body. Well,

1:26:21

actually, the elastic

1:26:23

fibers are

1:26:25

a very late development in

1:26:28

the evolution of life. And

1:26:34

these are structures that,

1:26:36

like rubber band, have

1:26:39

elasticity. And

1:26:41

these fibers are formed

1:26:43

in the first years

1:26:45

of the life. Around

1:26:48

puberty, no

1:26:51

more or only

1:26:53

very little elastic fibers

1:26:55

are formed anymore. So it's

1:26:57

a permanent structure. It's

1:26:59

very important for the

1:27:04

arteries, especially the main artery,

1:27:06

the outer, because

1:27:08

it gives elasticity. It

1:27:11

is important in the

1:27:13

lung because it gets

1:27:15

elasticity in breathing. And

1:27:17

it is also important

1:27:19

in the skin because

1:27:22

it gives the baby

1:27:24

face appearance of

1:27:26

the skin. And

1:27:28

if you get older, these

1:27:31

elastic fibers of the

1:27:34

skin are destroyed by

1:27:36

ultraviolet variation. So that's why

1:27:38

we look older when we get old.

1:27:42

But there have

1:27:44

been very

1:27:46

convincing reports that

1:27:48

people after the

1:27:50

vaccination suddenly appear

1:27:52

to look much older.

1:27:56

Now, this may be due to psychological

1:27:58

factors too, but We

1:28:01

definitely have proof that

1:28:03

these elastic fibers in

1:28:05

some cases are profoundly

1:28:07

destroyed in the skin. And

1:28:11

the other organ that

1:28:14

is very important in

1:28:16

view of elasticity is

1:28:18

the arteries. I mean

1:28:20

the heart contracts and

1:28:23

there's a rise in

1:28:25

pressure. And this pressure is

1:28:28

taken up by the elasticity.

1:28:30

So the blood

1:28:32

pressure is not going

1:28:34

up indefinitely, but

1:28:37

it is taken up. And

1:28:39

then when the heart is not

1:28:41

contracting, the arteries

1:28:44

flow the blood to

1:28:46

the organs. The

1:28:49

walls of the aorta and of

1:28:51

other major arteries are rich in

1:28:53

elastic fibers, which are arranged into

1:28:55

stacked layers or lamella. These

1:28:57

elastic lamella are essential for the

1:29:00

vessel's ability to withstand the pulsating

1:29:02

blood pressure. Professor

1:29:04

Burkhart found that in many of

1:29:06

his cases, the elastic lamella were

1:29:09

damaged and disrupted, particularly within the

1:29:11

hot spots of inflammation. If

1:29:14

the arteries are not

1:29:16

elastic, we would have

1:29:19

peaks in the blood pressure.

1:29:23

And the peaks, of course,

1:29:25

may lead to rupture. And

1:29:27

we already talked about rupture

1:29:30

of the arteries in

1:29:32

the brain in the aorta. Damage

1:29:35

to the aorta and to other

1:29:37

major arteries was also apparent in

1:29:39

patients who had not suffered overt

1:29:42

failure or rupture to these vessels.

1:29:45

This image shows the aortic wall

1:29:47

of a 29-year-old male who died

1:29:49

67 days after

1:29:51

receiving the second Pfizer injection. The

1:29:54

tissue sample has been treated with

1:29:56

a special stain, which highlights the disrupted

1:29:59

elastic lamella. The image on

1:30:01

the left shows intact elastic lamella

1:30:03

for a comparison. Can

1:30:05

you describe what we are viewing

1:30:07

here and the significant findings related

1:30:10

to the elastic fibers? Yes, well

1:30:12

on the left side you see that

1:30:15

the normal arteries, especially the

1:30:17

aorta, the main artery

1:30:19

of the body, is

1:30:21

constructed of a very

1:30:23

regular stratification of

1:30:27

myofabroplastic cells and smooth

1:30:29

muscle cells and these

1:30:32

elastic fibers. This is

1:30:34

very important and we

1:30:36

have very alarming findings.

1:30:40

First of all, destruction of elastic

1:30:42

fibers in the arteries,

1:30:45

especially in the aorta. Sometimes

1:30:48

very small lesions, you don't

1:30:50

see this in a radiograph.

1:30:53

Patients with these lesions

1:30:55

don't have any symptoms but those

1:30:58

that have further development which

1:31:00

have a total media

1:31:03

necrosis of the elastic fibers,

1:31:06

they may die

1:31:08

of aortic rupture and we have,

1:31:10

as I said, five cases of

1:31:12

this. Can you explain in simple

1:31:14

terms what media necrosis is or

1:31:17

how it would present itself? Well,

1:31:22

the larger arteries, especially the main

1:31:24

artery of the body, the aorta,

1:31:28

is made up of three

1:31:30

layers, the intima. This

1:31:34

is where the

1:31:36

arteriosclerosis and cholesterol

1:31:39

deposition happens. Then

1:31:42

we have the media, which

1:31:44

is where the

1:31:46

elastic fibers and the myofibroplastic,

1:31:48

the smooth muscle cells are

1:31:51

located. And then we

1:31:53

have the adventizia, which there

1:31:56

are the so-called vazavazurum,

1:31:58

which supply the

1:32:00

vessel ball with blood, oxygen

1:32:03

and so on. We have

1:32:05

the outside supplied by the

1:32:07

inside directly by perfusion. But

1:32:13

then we have the middle, the deep

1:32:15

media. And the deep media

1:32:18

is affected by toxic

1:32:20

agents and

1:32:22

by infectious toxic

1:32:24

agents. Now,

1:32:27

a hundred years ago, the

1:32:30

media necrosis

1:32:32

was very often seen in syphilis. It

1:32:38

was infectious, and it

1:32:41

also led to rupture

1:32:43

and death. And

1:32:47

this is probably because this

1:32:49

is what we call a

1:32:52

chilis heel of the otter,

1:32:55

where toxic agents, we act

1:32:57

there especially. And

1:33:01

there's also some kind

1:33:03

of food poisoning, which

1:33:06

is called latyrhism, which

1:33:10

by now we don't see very often. In

1:33:13

my first years as a pathologist, I

1:33:15

had a case where I saw

1:33:17

this. And it's

1:33:19

also a toxic agent in

1:33:21

some plants, Keche-Ebsen, a kind

1:33:24

of chickpeas, which may be

1:33:26

toxic. I suppose that this

1:33:28

is a phenomenon which is

1:33:30

similar to what we know have

1:33:33

seen in the past. So

1:33:35

there's a toxic and

1:33:37

maybe also immunologic attack in

1:33:40

the area of the arteries

1:33:42

where there's a weak point.

1:33:44

And there may be local

1:33:46

bleeding with hemocytorosis,

1:33:48

iron deposition deposit, and

1:33:51

there may be perforation.

1:33:54

And there may be probably in

1:33:58

many cases small... lesions

1:34:00

may heal. But then the elastic fibres

1:34:02

cannot be replaced by

1:34:04

elastic fibres once you

1:34:09

are older than 15 years, let's

1:34:12

say. And so there's a scar.

1:34:15

And if there's a scar,

1:34:17

the artery loses its elasticity

1:34:20

and so the blood pressure during contraction of

1:34:22

the heart is very high and it

1:34:24

goes down and it goes up and it goes down

1:34:26

and it leads to probably the

1:34:32

brain arteries are the most sensitive

1:34:34

arteries to rupture and death by terrible

1:34:41

bleeding. And now my fear

1:34:43

is maybe somebody who has a scar, a

1:34:46

scar, a scar, a scar, a scar in

1:34:54

his artery, maybe

1:34:56

he will die in five years from

1:34:58

terrible bleeding, but nobody will associate this

1:35:04

with the vaccination and nobody will even

1:35:06

examine the artery. This

1:35:08

is not a standard to

1:35:17

examine the artery. There will be

1:35:19

a high number of cases where

1:35:21

nobody sees any condition with

1:35:24

the vaccination, although it is probable.

1:35:27

As Professor Burkhart

1:35:29

mentioned earlier, damage to

1:35:34

elastic fibres was also commonly found

1:35:36

in the skin of vaccinated persons.

1:35:39

He observed these changes in

1:35:41

biopsies, that is, in

1:35:43

skin tissue samples of living patients.

1:35:45

Right now I'm

1:35:50

systematically reviewing our biopsies from the skin

1:35:52

and I have one example here very

1:36:00

delicate network of

1:36:02

very fine elastic fibers. They

1:36:04

are black. On the

1:36:06

top is the epiphyrium of

1:36:08

the dermis. And here you can see

1:36:10

this man, he's 38 years old and

1:36:14

he has a vasculitis

1:36:17

of the skin. Here you

1:36:19

can see there's these very

1:36:21

delicate black lines here.

1:36:24

These are the remnants of

1:36:26

the elastic fibers and there's

1:36:28

no network below

1:36:30

the basement membrane. Let's

1:36:34

now shift our focus back to clots.

1:36:36

And we did discuss this a little

1:36:38

bit earlier. And I think that the

1:36:40

topic of clots has received a lot

1:36:42

of public attention and has generated a

1:36:44

lot of public interest. I

1:36:46

want to first clarify before we discuss a

1:36:48

few images that there are

1:36:50

two types of clots associated with the

1:36:53

COVID-19 vaccines. Can you

1:36:55

explain those two clots? Well,

1:36:57

first of all, there's the, so

1:37:01

you may call normal thrombotic

1:37:03

clot which is

1:37:05

formed by thrombocytes and piperine.

1:37:08

And which, as I said

1:37:11

earlier, is of course a

1:37:16

kind of a healing of the traumatic

1:37:18

events. I mean, if you- So someone

1:37:20

gets a cut? If you cut your skin, of course,

1:37:25

there will be a thrombotic

1:37:28

formation and then the endothelium

1:37:30

regenerates and this is a

1:37:33

normal process. So that's one

1:37:35

type of clotting. That is when, for example,

1:37:37

somebody has a cut, it bleeds a little

1:37:39

bit, but eventually stops. That's one type and

1:37:41

that's considered normal. And the second type? Well,

1:37:44

the second time is a type

1:37:46

that has not been observed

1:37:48

before, actually. And

1:37:51

actually the first notice

1:37:53

of this came

1:37:55

from the United States. I

1:37:58

personally have been in the United States. And

1:38:00

I was a

1:38:02

guest for almost one year

1:38:05

with an undertaker. And

1:38:07

actually there I got my first experience

1:38:09

with dead people. I

1:38:12

know that from that time nobody

1:38:14

ever observed these

1:38:17

casts in the vessels.

1:38:20

Because in the

1:38:22

United States, different from Germany,

1:38:25

where people are buried or

1:38:27

burned, they embalm all deceased

1:38:30

persons are embalmed. And this makes

1:38:35

it necessary to open the

1:38:39

arteries or veins and

1:38:42

put taxation fluid into

1:38:44

the body. So the

1:38:46

body is embalmed. Soon

1:38:49

after this vaccination campaign

1:38:51

started, there were reports

1:38:53

from undertakers in the

1:38:55

United States that they

1:38:58

observed these very

1:39:01

strange casts

1:39:03

in the blood vessels. They

1:39:06

were long elastic

1:39:09

structures, not inherent

1:39:11

to the wall. So

1:39:13

they are not

1:39:15

caused by normal vascular damage.

1:39:19

And they

1:39:21

are very extensive. So just

1:39:24

from the first report that I read this,

1:39:26

I was convinced this

1:39:29

could not have been the

1:39:31

cause of the death. Because

1:39:34

if all your arteries are blocked,

1:39:36

you would die before all this

1:39:39

has formed. These clots

1:39:41

formed post-mortem, after death? I'm

1:39:43

absolutely sure they formed after

1:39:46

death. And they

1:39:48

were associated with the cooling of

1:39:51

the body. All

1:39:53

dead persons have to be cooled

1:39:55

before they are embalmed. These

1:39:58

are the two things that I... They

1:40:01

cannot have been formed in the

1:40:03

beta doing life

1:40:06

and they have been formed

1:40:08

by cooling. Now

1:40:12

we come to the point that we

1:40:14

have been observing

1:40:16

these phenomenon in living

1:40:18

persons. I think this

1:40:21

is something that has

1:40:24

not been looked into

1:40:26

before. In

1:40:29

cases in which abnormal blood clots

1:40:31

were observed in living patients, it

1:40:33

is important to note that the

1:40:35

clots were localized events and therefore

1:40:37

more survivable. In this case, the

1:40:39

patient is a woman in her

1:40:41

early 40s who was an avid

1:40:43

marathon runner. After receiving

1:40:46

one dose of the Pfizer

1:40:48

vaccine, the woman began to

1:40:50

experience blood perfusion problems and

1:40:52

sensitivity to cold temperatures. The

1:40:56

angiogram showed double-barreled arteries

1:40:58

in the legs. This

1:41:02

is the phenomenon that I

1:41:05

described before that the

1:41:07

media necrosis, in this case it

1:41:09

was not in the otter but

1:41:11

it was in the lower leg

1:41:14

arteries. This lady

1:41:16

was fortunate in a

1:41:18

way that this media

1:41:21

necrosis did not rupture

1:41:23

but it found its way back.

1:41:26

It is well known that there

1:41:28

are two ways. If

1:41:31

you have a dissection of the otter

1:41:34

or a large vessel, there are two

1:41:36

ways. Either it goes outside and you die

1:41:38

by bleeding, by

1:41:40

hemorrhage. The

1:41:45

other way is that it

1:41:47

finds its way back at

1:41:50

some other location and then

1:41:52

you have the circulation

1:41:55

again possible but

1:41:57

you have of course trouble with the

1:42:00

perfusion. And this

1:42:02

lady actually she was active

1:42:04

marathon runner. She was, she

1:42:07

participated in marathon runs and

1:42:10

soon after this vaccination

1:42:12

she could not walk anymore

1:42:14

for some time and had

1:42:16

very severe problems. She did

1:42:18

all kinds of therapeutic

1:42:21

measurements like plasmapheresis

1:42:24

and things like that and

1:42:26

she is better now but it

1:42:29

relapses. This is after the

1:42:31

vaccination that shows that there's a

1:42:33

profound damage of the perfusion. I

1:42:35

mean she at some times she

1:42:37

could not walk. She

1:42:40

couldn't walk. Walk anymore no. Wow. And

1:42:42

40 years old otherwise healthy.

1:42:45

Yes as I said a

1:42:47

marathon runner. Now this is

1:42:49

also a point of interest this image

1:42:51

here on the left which shows the

1:42:53

blood after it has been separated and

1:42:55

cooled. What is this yellow

1:42:58

structure that we see at the top of that

1:43:00

vial? Can you explain what's happening here? Yes

1:43:02

well let me first say that she had

1:43:06

a biopsy of the skin and

1:43:09

in the skin biopsy we saw

1:43:11

a vasculitis. We saw

1:43:14

necrosis of endothelium. We

1:43:17

saw expression of

1:43:19

spike protein and

1:43:22

then she called me and said well

1:43:25

the doctor took blood for

1:43:27

analysis, centrifuge it and

1:43:30

put it in the refrigerator and

1:43:33

the strange things happened that

1:43:37

in the upper

1:43:39

part where the serum is

1:43:41

they formed this strange clot

1:43:44

which apparently is not trumbous

1:43:47

because it's white as you see

1:43:49

it's there's no erythrocytes in there

1:43:52

and it is like

1:43:54

jellyfish and a little bit

1:43:56

sticky. Professor

1:43:59

Burkhart also examined the clot under

1:44:01

the microscope, which is shown here. He

1:44:04

used a special staining technique that highlights

1:44:06

fibrin. The extracellular proteins

1:44:09

within the clot were identified

1:44:11

with modern biochemical techniques in

1:44:13

another laboratory. We

1:44:15

found it was almost

1:44:19

cell-free aggregation of

1:44:22

small microfibrins,

1:44:26

probably un-mature fibrin.

1:44:30

The thing is, it's

1:44:32

definitely not a normal

1:44:34

thrombosis. We have fibrin,

1:44:36

which is a constituent

1:44:38

of a thrombus. It's

1:44:41

only on the surface. We

1:44:44

have inside these small fibrins.

1:44:49

This is the surface. You

1:44:51

see here, this is slightly

1:44:53

bluish. It's

1:44:56

very delicate fibers, which

1:44:59

probably are some

1:45:01

pre-stage fibrin. On

1:45:06

the surface, you see there is only

1:45:09

on the surface thrombocytes, some

1:45:11

lymphocytes. There is mature

1:45:13

fibrin on the surface,

1:45:16

which is most important. There

1:45:21

are CD61, which is a constituent

1:45:24

of endothelial cells. The

1:45:26

contents of endothelial cells comes

1:45:29

into the blood. Under

1:45:33

certain circumstances, apparently after

1:45:35

cooling, they

1:45:38

form these structures,

1:45:41

these clots. Then we

1:45:44

had protanomic analysis done

1:45:46

by a friendly

1:45:49

laboratory, and they found

1:45:52

that the protein composition

1:45:55

of the serum and of the

1:45:57

clot differed. And

1:46:00

in the clot, there

1:46:02

were 139 protein

1:46:05

structures that were not in the

1:46:07

serum. And

1:46:09

these were exocilia

1:46:11

lematrix, colargain, kylostine,

1:46:13

and some other

1:46:16

structures, especially CD31,

1:46:18

which is related

1:46:20

to endothelial contents.

1:46:24

We consider now,

1:46:26

and of course we have more

1:46:29

cases to examine. We have some

1:46:31

more already now. We

1:46:34

concluded that these

1:46:37

clot formations are an

1:46:40

indication that in

1:46:42

the past there was

1:46:44

an endothelial damage. And

1:46:48

if it's still forming, it's an ongoing

1:46:52

endothelial damage. And through

1:46:55

the endothelial damage, proteins

1:46:57

and matrix constituents of the

1:47:00

vessel ball come into the

1:47:02

blood and circulate in the

1:47:04

blood. And

1:47:06

under certain circumstances, they

1:47:09

can form these clots.

1:47:12

And you were seeing this in several

1:47:14

people who were vaccinated, these types of

1:47:16

clots. Is that correct? Yes. Okay.

1:47:20

But at this moment, we

1:47:22

don't have a systematic evaluation,

1:47:24

but it seems to

1:47:26

be the case that it

1:47:29

is associated with vaccination. In

1:47:32

all of your years of pathology prior to

1:47:34

the rollout of the COVID-19 vaccinations, you did

1:47:36

not see this type of blood clotting. Is

1:47:39

that correct? That is correct, yes.

1:47:42

And actually we have one

1:47:46

specimen which was

1:47:50

taken from a person who was

1:47:52

still living. And they did angioplasty

1:47:55

and they removed such a

1:47:57

clot outside of the artery.

1:48:01

And this is definitely associated

1:48:03

with temperature because

1:48:07

this lady especially, but I have

1:48:09

heard it also from other persons,

1:48:11

they feel or they have no

1:48:14

major problems as long as the

1:48:16

temperature is good. But

1:48:20

if it's below 25 degrees,

1:48:22

they have problems

1:48:24

with their hands

1:48:27

maybe. Is

1:48:29

it their circulation? Apparently. They

1:48:32

have problems in the cold temperature and

1:48:34

multiple people who have been vaccinated are reporting

1:48:36

this? Yes. Okay. Next,

1:48:40

Professor Burkhardt and I discuss his

1:48:42

findings and his work more generally.

1:48:45

He provides his perspective on the

1:48:48

scientific community, academic science and

1:48:50

the public health industry. He

1:48:52

also reflects back on his

1:48:54

career and shares his motivation

1:48:57

for doing this work. All

1:49:00

right, Professor Burkhardt, I just want to

1:49:02

have a general discussion with you about

1:49:05

the findings, about your work. And

1:49:07

first of all, I would just like to know to

1:49:09

what degree of certainty are you

1:49:11

able to show or that you can

1:49:13

say that the damage that you have

1:49:16

observed is associated with the COVID-19 vaccines?

1:49:21

Well, as I said, there's

1:49:24

not a single test or a single

1:49:28

histological change or immunohistological

1:49:30

change. It's always the

1:49:33

combination of all

1:49:35

the findings that we see. And of course,

1:49:37

we have to take into account the

1:49:40

medical history. In the 75 cases now,

1:49:42

we have 78% where we are certain

1:49:44

that the

1:49:46

test process was in some way

1:49:58

influenced by the vaccination. This

1:50:00

does not say that all these

1:50:03

people died of the vaccination. The

1:50:06

death process is complicated. In

1:50:09

persons over 50 years, of course,

1:50:11

there are many

1:50:15